Amyloid Beta Mouse Monoclonal Antibody - Purified | FR | MédiMabs

Amyloid Beta Mouse Monoclonal Antibody - Purified

Amyloid Beta Mouse Monoclonal Antibody - Purified

Amyloid Beta Mouse Monoclonal Antibody - Purified

MM-0015-P -- anti-Amyloid Beta
$482,00/100 µg
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NUMÉRO : MM-0015-P
  • Nom du produit
    Amyloid Beta Mouse Monoclonal Antibody - Purified
  • Serveur
    Mouse
  • Clonality
    Monoclonal
  • Tested Applications
    Immunohistochemistry (IHC), Electron microscopy immunocytochemistry (ICC), Immunoprecipitation (IP)
  • Research Area(s)
    Neuroscience
  • Immunogen
    Full lenght Amyloid beta
  • Modifications
    None
  • Clone ID
    McSA1
  • DataSHEET
  • Isotype
    IgG1
  • Conjugation
    None
  • Reactivity
    Both neuritic and diffuse plaques can be detected as well as cerebrovascular amyloid in affected cortex of Alzheimer’s disease (AD) brain by ICC
  • Specificity
    Human and Mouse
  • Purification
    Protein G
  • Target Name
    Amyloid Beta
  • Storage Buffer
    PBS
  • Alias
    Beta-amyloid ABP amyloid peptide
  • Format
    100 µg
  • Background
    Progressive deposition of insoluble aggregates of the 39 to 43 amino acid amyloid β (Aβ) peptide derived from the proteolytic cleavage of the amyloid β-protein precursor (AβPP), gives rise to one of the pathological hallmarks of Alzheimer’s disease (AD). The formation of Aβ fragments is a critical determinant in unleashing AD neuropathology.

For laboratory research only, not for drug, diagnostic or other use.

  • Reelin-immunoreactive neurons in entorhinal cortex layer II selectively express intracellular amyloid in early Alzheimer's disease
  • Intraneuronal Amyloid Beta Accumulation Disrupts Hippocampal CRTC1Dependent Gene Expression and Cognitive Function in a Rat Model of Alzheimer Disease
  • Stereological estimation of neuron number and plaque load in the hippocampal region of a transgenic rat model of Alzheimer’s disease
  • The Multi-Target Drug M30 Shows Pro-Cognitive and Anti-Inflammatory Effects in a Rat Model of Alzheimer’s Disease
  • Longitudinal testing of hippocampal plasticity reveals the onset and maintenance of endogenous human Aß-induced synaptic dysfunction in individual freely behaving pre-plaque transgenic rats: rapid reversal by anti-Aß agents
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